We reported very recently that e-cigarettes are detrimental to mouth health. According to new research from the University of Rochester Medical Center, e-cigarettes cause damage to gum tissue.
With all the attention focused on cigarettes, other products and substitutions have slipped through the cracks. One product in particular is becoming increasingly popular: E-cigarettes.
E-cigarettes use atomizers to create a vapor by heating up artificial juices and flavorings. Because there is no smoke, just vapor, people have assumed that they are safe.
The study was led by Irfan Rahman, Ph.D. professor of Environmental Medicine at the UR School of Medicine and Dentistry, and is the first scientific study to examine the effects of e-cigarettes on oral health.
“We showed that when the vapors from an e-cigarette are burned, it causes cells to release inflammatory proteins, which in turn aggravate stress within cells, resulting in damage that could lead to various oral diseases,” explained Rahman. “How much and how often someone is smoking e-cigarettes will determine the extent of damage to the gums and oral cavity.”
The study also found that the chemicals that create flavors in the liquid play a part in damaging mouth cells.
“We learned that the flavorings-some more than others — made the damage to the cells even worse,” added Fawad Javed, a post-doctoral resident at Eastman Institute for Oral Health, part of the UR Medical Center, who contributed to the study. “It’s important to remember that e-cigarettes contain nicotine, which is known to contribute to gum disease.”
“More research, including long term and comparative studies, are needed to better understand the health effects of e-cigarettes,” added Rahman.
The study showing that e-cigarettes cause damage to gum tissue was published in the journal Oncotarget.
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REFERENCES:
1. “First-ever Study Shows E-cigarettes Cause Damage to Gum Tissue.” University of Rochester Medical Center. University of Rochester Medical Center, n.d. Web. 26 Nov. 2016.
2. “E-cigarettes and Flavorings Induce Inflammatory and Pro-senescence Responses in Oral Epithelial Cells and Periodontal Fibroblasts.” Oncotarget. Oncotarget, n.d. Web. 26 Nov. 2016.
